Loss of Function of Slc20a2 Associated with Familial Idiopathic Basal Ganglia Calcification in Humans Causes Brain Calcifications in Mice-Reference-Cited by-同舟云学术

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Loss of Function of Slc20a2 Associated with Familial Idiopathic Basal Ganglia Calcification in Humans Causes Brain Calcifications in Mice

Published:2013-08-10 Issue:3 Volume:51 Page:994-999
ISSN:0895-8696
Container-title:Journal of Molecular Neuroscience
language:en
Short-container-title:J Mol Neurosci

Author:

Jensen Nina,Schrøder Henrik Daa,Hejbøl Eva Kildall,Füchtbauer Ernst-Martin,de Oliveira João Ricardo Mendes,Pedersen Lene

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s12031-013-0085-6.pdf

Reference22 articles.

1. Bøttger P, Pedersen L (2002) Two highly conserved glutamate residues critical for type III sodium-dependent phosphate transport revealed by uncoupling transport function from retroviral receptor function. J Biol Chem 277:42741–47

2. Bøttger P, Pedersen L (2011) Mapping of the minimal inorganic phosphate transporting unit of human PiT2 suggests a structure universal to PiT-related proteins from all kingdoms of life. BMC Biochem 12:21

3. Bøttger P, Hede SE, Grunnet M, Høyer B, Klaerke DA, Pedersen L (2006) Characterization of transport mechanisms and determinants critical for Na+-dependent Pi symport of the PiT-family paralogs, human PiT1 and PiT2. Am J Physiol Cell Physiol 291:C1377–87

4. Byskov K, Jensen N, Kongsfelt IB, Wielsøe M, Pedersen LE, Haldrup H et al (2012) Regulation of cell proliferation and cell density by the inorganic phosphate transporter PiT1. Cell Div 7:7

5. Covic A, Kothawala P, Bernal M, Robbins S, Chalian A, Goldsmith D (2009) Systematic review of the evidence underlying the association between mineral metabolism disturbances and risk of all-cause mortality, cardiovascular mortality and cardiovascular events in chronic kidney disease. Nephrol Dial Transplant 24:1506–23

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