WT1 Alternative Splicing: Role of Its Isoforms in Neuroblastoma
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,General Medicine
Link
http://link.springer.com/article/10.1007/s12031-017-0930-0/fulltext.html
Reference109 articles.
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2. Armstrong JF, Pritchard-Jones K, Bickmore WA, Hastie ND, Bard JB (1993) The expression of the Wilms’ tumour gene, WT1, in the developing mammalian embryo. Mech Dev 40:85–97
3. Avigad S, Feinberg-Gorenshtein G, Luria D, Jeison M, Stein J, Grunshpan A, Sverdlov Y, Ash S, Yaniv I (2009) Minimal residual disease in peripheral blood stem cell harvests from high-risk neuroblastoma patients. J Pediatr Hematol Oncol 31:22–26
4. Bansal H, Seifert T, Bachier C, Rao M, Tomlinson G, Iyer SP, Bansal S (2012) The transcription factor Wilms tumor 1 confers resistance in myeloid leukemia cells against the proapoptotic therapeutic agent TRAIL (tumor necrosis factor α-related apoptosis-inducing ligand) by regulating the antiapoptotic protein Bcl-xL. J Biol Chem 287:32875–32880
5. Barbaux S, Niaudet P, Gubler MC, Grünfeld JP, Jaubert F, Kuttenn F, Fékété CN, Souleyreau-Therville N, Thibaud E, Fellous M, McElreavey K (1997) Donor splice-site mutations in WT1 are responsible for Frasier syndrome. Nat Genet 17:467–470
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