Comparison of Pharmacological Modulation of APP Metabolism in Primary Chicken Telencephalic Neurons and in a Human Neuroglioma Cell Line-Reference-Cited by-同舟云学术

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Comparison of Pharmacological Modulation of APP Metabolism in Primary Chicken Telencephalic Neurons and in a Human Neuroglioma Cell Line

Published:2010-07-06 Issue:3 Volume:43 Page:257-267
ISSN:0895-8696
Container-title:Journal of Molecular Neuroscience
language:en
Short-container-title:J Mol Neurosci

Author:

Czvitkovich Stefan,Duller Stephan,Mathiesen Else,Lorenzoni Klaus,Imbimbo Bruno P.,Hutter-Paier Birgit,Windisch Manfred,Wronski Robert

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine

Link

http://www.springerlink.com/index/pdf/10.1007/s12031-010-9416-z

Reference49 articles.

1. Carrodeguas JA, Rodolosse A, Garza MV et al (2005) The chick embryo appears as a natural model for research in beta-amyloid precursor protein processing. Neuroscience 134:1285–1300

2. Citron M, Oltersdorf T, Haass C et al (1992) Mutation of the beta-amyloid precursor protein in familial Alzheimer's disease increases beta-protein production. Nature 360:672–674

3. Citron M, Teplow DB, Selkoe DJ (1995) Generation of amyloid beta protein from its precursor is sequence specific. Neuron 14:661–670

4. Cole SL, Vassar R (2008) The role of amyloid precursor protein processing by BACE1, the beta-secretase, in Alzheimer disease pathophysiology. J Biol Chem 283:29621–29625

5. Czirr E, Leuchtenberger S, Dorner-Ciossek C et al (2007) Insensitivity to Abeta42-lowering nonsteroidal anti-inflammatory drugs and gamma-secretase inhibitors is common among aggressive presenilin-1 mutations. J Biol Chem 282:24504–24513

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