Molecular Mechanisms of Epilepsy: The Role of the Chloride Transporter KCC2
Author:
Funder
FCT – Fundação para a Ciência e a Tecnologia
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s12031-022-02041-7.pdf
Reference183 articles.
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2. Akerman CJ, Cline HT (2006) Depolarizing GABAergic conductances regulate the balance of excitation to inhibition in the developing retinotectal circuit in vivo. J Neurosci 26(19):5117–5130. https://doi.org/10.1523/JNEUROSCI.0319-06.2006
3. Akyuz E, Polat AK, Eroglu E, Kullu I, Angelopoulou E, Paudel YN (2021) Revisiting the role of neurotransmitters in epilepsy: an updated review. Life Sci 265:118826. https://doi.org/10.1016/j.lfs.2020.118826
4. Albertson AJ, Yang J, Hablitz JJ (2011) Decreased hyperpolarization-activated currents in layer 5 pyramidal neurons enhances excitability in focal cortical dysplasia. J Neurophysiol 106(5):2189–2200. https://doi.org/10.1152/jn.00164.2011
5. Anstotz M, Fiske MP, Maccaferri G (2021) Impaired KCC2 Function Triggers interictal-like activity driven by parvalbumin-expressing interneurons in the isolated subiculum in vitro. Cereb Cortex 31(10):4681–4698. https://doi.org/10.1093/cercor/bhab115
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