Contributions of prolonged contingent and non-contingent cocaine exposure to escalation of cocaine intake and glutamatergic gene expression-Reference-Cited by-同舟云学术

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Contributions of prolonged contingent and non-contingent cocaine exposure to escalation of cocaine intake and glutamatergic gene expression

Published:2017-12-12 Issue:5 Volume:235 Page:1347-1359
ISSN:0033-3158
Container-title:Psychopharmacology
language:en
Short-container-title:Psychopharmacology

Author:

Ploense Kyle L.,Vieira Philip,Bubalo Lana,Olivarria Gema,Carr Amanda E.,Szumlinski Karen K.,Kippin Tod E.

Funder

National Institute on Drug Abuse

W. M. Keck Foundation

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology

Link

http://link.springer.com/article/10.1007/s00213-017-4798-z/fulltext.html

Reference78 articles.

1. Ahmed S, Cador M (2006) Dissociation of psychomotor sensitization from compulsive cocaine consumption. Neuropsychopharmacology 31:9

2. Ahmed S, Koob GF (1998) Transition from moderate to excessive drug intake: change in hedonic set point. Science 282(5387):298–300. https://doi.org/10.1126/science.282.5387.298

3. Ahmed SH, Kenny PJ, Koob GF, Markou A (2002) Neurobiological evidence for hedonic allostasis associated with escalating cocaine use. Nat Neurosci 5(7):625–626. https://doi.org/10.1038/nn872

4. Ahmed SH, Koob GF (1999) Long-lasting increase in the set point for cocaine self-administration after escalation in rats. Psychopharmacology 146(3):303–312. https://doi.org/10.1007/s002130051121

5. Ahmed SH, Koob GF (2005) Transition to drug addiction: a negative reinforcement model based on an allostatic decrease in reward function. Psychopharmacology 180(3):473–490. https://doi.org/10.1007/s00213-005-2180-z

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