The hindbrain is a site of energy balance action for prolactin-releasing peptide: feeding and thermic effects from GPR10 stimulation of the nucleus tractus solitarius/area postrema
Author:
Funder
National Institutes of Health
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology
Link
http://link.springer.com/article/10.1007/s00213-018-4925-5/fulltext.html
Reference63 articles.
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2. Alhadeff AL, Mergler BD, Zimmer DJ, Turner CA, Reiner DJ, Schmidt HD, Grill HJ, Hayes MR (2017) Endogenous glucagon-like peptide-1 receptor signaling in the nucleus tractus solitarius is required for food intake control. Neuropsychopharmacology 42:1471–1479. https://doi.org/10.1038/npp.2016.246
3. Bechtold DA, Luckman SM (2006) Prolactin-releasing peptide mediates cholecystokinin-induced satiety in mice. Endocrinology 147:4723–4729. https://doi.org/10.1210/en.2006-0753
4. Bjursell M, Lennerås M, Göransson M, Elmgren A, Bohlooly-Y M (2007) GPR10 deficiency in mice results in altered energy expenditure and obesity. Biochem Biophys Res Commun 363:633–638. https://doi.org/10.1016/j.bbrc.2007.09.016
5. Chen CT, Dun SL, Dun NJ, Chang JK (1999) Prolactin-releasing peptide-immunoreactivity in A1 and A2 noradrenergic neurons of the rat medulla. Brain Res 822:276–279. https://doi.org/10.1016/S0006-8993(99)01153-1
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