Cannabinoid receptor GPR55 activation blocks nicotine use disorder by regulation of AMPAR phosphorylation-Reference-Cited by-同舟云学术

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Cannabinoid receptor GPR55 activation blocks nicotine use disorder by regulation of AMPAR phosphorylation

Published:2021-10-14 Issue:11 Volume:238 Page:3335-3346
ISSN:0033-3158
Container-title:Psychopharmacology
language:en
Short-container-title:Psychopharmacology

Author:

Liu Qingqing,Yu Jiaoyan,Li Xi,Guo Yanyan,Sun Ting,Luo Li,Ren Jing,Jiang Wei,Zhang Ruitao,Yang Peng,Yang Qi

Funder

National Natural Science Foundation of China

National Postdoctoral Program for Innovative Talents

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology

Link

https://link.springer.com/content/pdf/10.1007/s00213-021-05949-x.pdf

Reference34 articles.

1. Alavi MS, Hosseinzadeh H, Shamsizadeh A, Roohbakhsh A (2016) The effect of O-1602, an atypical cannabinoid, on morphine-induced conditioned place preference and physical dependence. Pharmacological Reports : PR 68:592–597

2. Ashare RL, Kimmey BA, Rupprecht LE, Bowers ME, Hayes MR, Schmidt HD (2016) Repeated administration of an acetylcholinesterase inhibitor attenuates nicotine taking in rats and smoking behavior in human smokers. Transl Psychiatr 6: e713

3. Benowitz NL (2010) Nicotine addiction. N Engl J Med 362:2295–2303

4. Briand LA, Deutschmann AU, Ellis AS, Fosnocht AQ (2016) Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking. Neuropharmacology 111:231–241

5. Collaborators GBDT (2017) Smoking prevalence and attributable disease burden in 195 countries and territories, 1990–2015: a systematic analysis from the Global Burden of Disease Study 2015. Lancet 389:1885–1906

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