IL4Rα and IL17A Blockade Rescue Autoinflammation in SOCS1 Haploinsufficiency
Author:
Funder
National Institute of Allergy and Infectious Diseases
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
https://link.springer.com/content/pdf/10.1007/s10875-023-01635-z.pdf
Reference27 articles.
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3. Meuwissen ME, et al. Human USP18 deficiency underlies type 1 interferonopathy leading to severe pseudo-TORCH syndrome. J Exp Med. 2016;213(7):1163–74.
4. Gruber C, et al. Homozygous STAT2 gain-of-function mutation by loss of USP18 activity in a patient with type I interferonopathy. J Exp Med. 2020;217(5)
5. Zhang X, et al. Human intracellular ISG15 prevents interferon-alpha/beta over-amplification and auto-inflammation. Nature. 2015;517(7532):89–93.
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1. SOCS1 is a critical checkpoint in immune homeostasis, inflammation and tumor immunity;Frontiers in Immunology;2024-06-14
2. One gene to rule them all – clinical perspectives of a potent suppressor of cytokine signaling – SOCS1;Frontiers in Immunology;2024-04-22
3. Mendelian Causes of Autoimmunity: the Lupus Phenotype;Journal of Clinical Immunology;2024-04-15
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