Depletion of regulatory T cells by anti-GITR mAb as a novel mechanism for cancer immunotherapy-Reference-Cited by-同舟云学术

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Depletion of regulatory T cells by anti-GITR mAb as a novel mechanism for cancer immunotherapy

Published:2010-05-18 Issue:9 Volume:59 Page:1367-1377
ISSN:0340-7004
Container-title:Cancer Immunology, Immunotherapy
language:en
Short-container-title:Cancer Immunol Immunother

Author:

Coe David,Begom Shaima,Addey Caroline,White Matthew,Dyson Julian,Chai Jian-Guo

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Oncology,Immunology,Immunology and Allergy

Link

http://link.springer.com/content/pdf/10.1007/s00262-010-0866-5.pdf

Reference44 articles.

1. Nocentini G, Giunchi L, Ronchetti S et al (1997) A new member of the tumor necrosis factor/nerve growth factor receptor family inhibits T cell receptor-induced apoptosis. Proc Natl Acad Sci USA 94:6216–6221

2. Gurney AL, Marsters SA, Huang RM et al (1999) Identification of a new member of the tumor necrosis factor family and its receptor, a human ortholog of mouse GITR. Curr Biol 9:215–218

3. Kwon B, Yu KY, Ni J et al (1999) Identification of a novel activation-inducible protein of the tumor necrosis factor receptor superfamily and its ligand. J Biol Chem 274:6056–6061

4. Shimizu J, Yamazaki S, Takahashi T et al (2002) Stimulation of CD25(+)CD4(+) regulatory T cells through GITR breaks immunological self-tolerance. Nat Immunol 3:135–142

5. McHugh RS, Whitters MJ, Piccirillo CA et al (2002) CD4+ CD25+ immunoregulatory T cells: gene expression analysis reveals a functional role for the glucocorticoid-induced TNF receptor. Immunity 16:311–323

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