Streptozotocin-induced diabetes is not a model for methylmalonic acidaemia
Author:
Publisher
Wiley
Subject
Genetics(clinical),Genetics
Link
http://www.springerlink.com/index/pdf/10.1007/BF01802038
Reference20 articles.
1. Asunta, J., Koivusalo, M. and Suorsa, M. Excretion of methylmalonate and succinate in the urine of rats during deficiency of vitamin B12 and after administration of propionate.Acta Physiol. Scand. 66 (1966) 346–351
2. Bartels, H., Böhmer, M. and Heierli, C. Serum creatinine determination without deproteination.Clin. Chim. Acta 37 (1972) 193–197
3. Bhatt, H. R. and Linnell, J. C. Induction of ethylmalonic acidaemia with streptozotocin: a possible experimental model for the study of methylmalonic acidaemia.J. Inher. Metab. Dis. 6 Suppl. 2 (1983) 119–120
4. Bhatt, H. R., Green, A. and Linnell, J. C. A sensitive micromethod for the routine estimation of methylmalonic acid in body fluids and tissues using thin layer chromatography.Clin. Chim. Acta 18 (1982) 311–321
5. Bhatt, H. R., Linnell, J. C. and Matthews, D. M. Can faulty vitamin B12 (Cobalamin) metabolism produce diabetic neuropathy.Lancet Sept. 3 (1983) 572
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1. Disorders of cobalamin (Vitamin B12) metabolism: Emerging concepts in pathophysiology, diagnosis and treatment;Blood Reviews;2007-05
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