Erythromelalgia
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11936-006-0008-8.pdf
Reference70 articles.
1. Mork C, Asker CL, Salerud EG, Kvernebo K: Microvascular arteriovenous shunting is a probable pathogenetic mechanism in erythromelalgia. J Invest Dermatol 2000, 114:643–646.
2. Davis MD, Rooke TW, Sandroni P: Mechanisms other than shunting are likely contributing to the pathophysiology of erythromelalgia. J Invest Dermatol 2000, 115:1166–1167.
3. Davis M, Sandroni P, Harper CM, et al.: Neurophysiologic and vascular studies in erythromelalgia: a retrospective analysis. J Clin Neuromuscular Dis 1999, 1:57–63.
4. Drenth JP, te Morsche RH, Guillet G, et al.: SCN9A mutations define primary erythermalgia as a neuropathic disorder of voltage gated sodium channels. J Invest Dermatol 2005, 124:1333–1338.
5. Davis MD, O’Fallon WM, Rogers RS 3rd, Rooke TW: Natural history of erythromelalgia: presentation and outcome in 168 patients. Arch Dermatol 2000, 136:330–336. This paper reviews the presentation, natural history, and prognosis in 168 patients with erythromelalgia. In addition, a survey of patients documents quality-of-life scores and tabulates their self-reported response to treatment.
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