SIRT1 Activation Promotes Long-Term Functional Recovery After Subarachnoid Hemorrhage in Rats
Author:
Funder
NIH
Publisher
Springer Science and Business Media LLC
Subject
Critical Care and Intensive Care Medicine,Neurology (clinical)
Link
https://link.springer.com/content/pdf/10.1007/s12028-022-01614-z.pdf
Reference64 articles.
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2. Pillarisetti S. A review of Sirt1 and Sirt1 modulators in cardiovascular and metabolic diseases. Recent Pat Cardiovasc Drug Discov. 2008;3:156–64.
3. Blander G, Guarente L. The Sir2 family of protein deacetylases. Annu Rev Biochem. 2004;73:417–35.
4. Dryden SC, Nahhas FA, Nowak JE, Goustin AS, Tainsky MA. Role for human SIRT2 NAD-dependent deacetylase activity in control of mitotic exit in the cell cycle. Mol Cell Biol. 2003;23:3173–85.
5. Grimm AA, Brace CS, Wang T, Stormo GD, Imai S. A nutrient-sensitive interaction between Sirt1 and HNF-1alpha regulates Crp expression. Aging Cell. 2011;10:305–17.
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1. NAD-Dependent Protein Deacetylase Sirtuin-1 Mediated Mitophagy Regulates Early Brain Injury After Subarachnoid Hemorrhage;Journal of Inflammation Research;2024-03
2. Dl-3-n-Butylphthalide attenuates early brain injury and delayed neurological dysfunction by regulating NLRP3 inflammasome after subarachnoid hemorrhage;2023-11-20
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