Biochemical and behavioral phenotype of AGAT and GAMT deficient mice following long-term Creatine monohydrate supplementation-Reference-Cited by-同舟云学术

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Biochemical and behavioral phenotype of AGAT and GAMT deficient mice following long-term Creatine monohydrate supplementation

Published:2017-08-14 Issue:6 Volume:32 Page:1951-1961
ISSN:0885-7490
Container-title:Metabolic Brain Disease
language:en
Short-container-title:Metab Brain Dis

Author:

Iqbal Furhan,Hoeger Herald,Lubec Gurt,Bodamer Olaf

Funder

Higher Education Commission, Pakistan

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Biochemistry

Link

http://link.springer.com/article/10.1007/s11011-017-0092-3/fulltext.html

Reference36 articles.

1. AlmediaL S, Vilarinho L, Darmin PS, Rosenberg EH, Martinez-Munoz C, Jakobs C et al (2007) A prevalent pathogenic GAMT mutation (c.59G>C) in Portugal. Mol Genet Metab 91:1–6

2. Almeida LS, Salomons GS, Hogenboom F, Jakobs C, Schoffelmeer AN (2006) Exocytotic release of creatine in rat brain. Synapse 60(2):118–123

3. Amital D, Vishne T, Roitman S, Kotler M, Levine J (2006) Open study of creatinemonohydrate in treatment-resistant posttraumatic stress disorder. J Clin Psychiat 67:836–837

4. Battini B, Euzzi V, Carducci C, Tosetti M, Binachi MC, Item CB et al (2002) Creatine depletion in a new case with AGAT deficiency: clinical and genetic study in a large pedigree. Mol Gen Metab 77:326–331

5. Bianchi MC, Tosetti M, Fornai F, Alessandri MG, Cipriani P, De Vito G et al (2000) Reversible brain creatine deficiency in two sisters with normal blood creatine level. Ann Neurol 47:511–513

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