In schizophrenia, non-remitters and partial remitters to treatment with antipsychotics are qualitatively distinct classes with respect to neurocognitive deficits and neuro-immune biomarkers: results of soft independent modeling of class analogy-Reference-Cited by-同舟云学术

In schizophrenia, non-remitters and partial remitters to treatment with antipsychotics are qualitatively distinct classes with respect to neurocognitive deficits and neuro-immune biomarkers: results of soft independent modeling of class analogy

Published:2021-02-13 Issue:5 Volume:36 Page:939-955
ISSN:0885-7490
Container-title:Metabolic Brain Disease
language:en
Short-container-title:Metab Brain Dis

Author:

Al-Hakeim Hussein Kadhem,Mousa Rana Fadhil,Al-Dujaili Arafat Hussein,Maes Michael^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Biochemistry

Link

https://link.springer.com/content/pdf/10.1007/s11011-021-00685-9.pdf

Reference69 articles.

1. Al-Dujaili, AH, Mousa RH, Al-Hakeim HK, Maes M (2020) 'High Mobility Group Protein 1 and Dickkopf-Related Protein 1 in Schizophrenia and Treatment-Resistant Schizophrenia: Associations with Interleukin-6, Symptom Domains, and Neurocognitive Impairments'. Schizophr Bull.sbaa136. doi: 10.1093/schbul/sbaa136

2. Al-Hakeim HK, Almulla AF, Al-Dujaili AH, Maes M (2020a) Construction of a neuro-immune-cognitive pathway-phenotype underpinning the phenome of deficit schizophrenia. Curr Top Med Chem, in press 20:747–758

3. Al-Hakeim HK, Almulla AF, Maes M (2020b) The Neuroimmune and neurotoxic fingerprint of Major neurocognitive psychosis or deficit schizophrenia: a supervised machine learning study. Neurotox Res 37:753–771

4. Almulla AF, Al-Hakeim HK, Maes M (2020) Schizophrenia phenomenology revisited: positive and negative symptoms are strongly related reflective manifestations of an underlying single trait indicating overall severity of schizophrenia. CNS Spectr 20:1–10

5. Andersson U, Tracey KJ (2011) HMGB1 is a therapeutic target for sterile inflammation and infection. Annu Rev Immunol 29:139–162

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