Interaction between CTLA4 gene and IBD5 locus in Hungarian Crohn’s disease patients-Reference-Cited by-同舟云学术

Interaction between CTLA4 gene and IBD5 locus in Hungarian Crohn’s disease patients

Published:2011-04-26 Issue:9 Volume:26 Page:1119-1125
ISSN:0179-1958
Container-title:International Journal of Colorectal Disease
language:en
Short-container-title:Int J Colorectal Dis

Author:

Csöngei Veronika,Járomi Luca,Sáfrány Enikő,Sipeky Csilla,Magyari Lili,Polgár Noémi,Bene Judit,Sarlós Patrícia,Lakner Lilla,Baricza Eszter,Szabó Melinda,Rappai Gábor,Melegh Béla

Publisher

Springer Science and Business Media LLC

Subject

Gastroenterology

Link

http://link.springer.com/content/pdf/10.1007/s00384-011-1202-z.pdf

Reference70 articles.

1. Moore JH (2003) The ubiquitous nature of epistasis in determining susceptibility to common human diseases. Hum Hered 56:73–82

2. Cordell HJ (2002) Epistasis: what it means, what it doesn’t mean, and statistical methods to detect it in humans. Hum Mol Genet 11:2463–2468

3. Magyari L, Farago B, Bene J et al (2007) No association of the cytotoxic T-lymphocyte associated gene CTLA4 +49A/G polymorphisms with Crohn’s disease and ulcerative colitis in Hungarian population samples. World J Gastroenterol 13:2205–2208

4. Talian G, Lakner L, Bene J et al (2009) Plasma carnitine ester profiles in Crohn’s disease and ulcerative colitis patients with different IGR2230a_1 genotypes. Int J Immunogenet 36:329–335

5. Lakner L, Csongei V, Sarlos P et al (2009) IGR2096a_1 T and IGR2198a_1 C alleles on IBD5 locus of chromosome 5q31 region confer risk for Crohn’s disease in Hungarian patients. Int J Colorectal Dis 24:503–507

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. CTLA-4 (+49A/G) and NOD2/CARD15 (N852S) polymorphisms with inflammatory bowel disease in Turkish patients;Cellular and Molecular Biology;2018-08-30

2. CTLA-4 Gene +49 A/G Polymorphism in Prostate Cancer Patients in Turkish Population;Advances in Cytology & Pathology;2017-10-30

3. Association of CTLA-4 variants with susceptibility to inflammatory bowel disease: A meta-analysis;Human Immunology;2014-03

4. Susceptibility to ulcerative colitis in Hungarian patients determined by gene-gene interactions;World Journal of Gastroenterology;2014