Upregulating the Expression of Survivin-HBXIP Complex Contributes to the Protective Role of IMM-H004 in Transient Global Cerebral Ischemia/Reperfusion-Reference-Cited by-同舟云学术

Upregulating the Expression of Survivin-HBXIP Complex Contributes to the Protective Role of IMM-H004 in Transient Global Cerebral Ischemia/Reperfusion

Published:2016-01-07 Issue:1 Volume:54 Page:524-540
ISSN:0893-7648
Container-title:Molecular Neurobiology
language:en
Short-container-title:Mol Neurobiol

Author:

Chu Shi-Feng,Zhang Zhao,Zhang Wei,Zhang Mei-Jin,Gao Yan,Han Ning,Zuo Wei,Huang Hui-Yong,Chen Nai-Hong

Funder

National Natural Science Foundation of China

the National Key Sci-Tech Major Special Item

the National High-Tech R&D Program (863 Program)

the Program for Changjiang Scholars and Innovative Research Team in University (PCSIRT)

the Specialized Research Fund for the Doctoral Program of Higher Education of China

Natural Science Foundation of Beijing Municipality (CN)

Beijing Key Laboratory of New Drug Mechanisms and Pharmacological Evaluation Study

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology

Link

http://link.springer.com/content/pdf/10.1007/s12035-015-9673-5.pdf

Reference33 articles.

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2. Li Y, Xia ZL, Chen LB (2011) HIF-1-alpha and survivin involved in the anti-apoptotic effect of 2ME2 after global ischemia in rats. Neurol Res 33:583–592

3. Ji HJ, Wang DM, Hu JF, Sun MN, Li G et al (2014) IMM-H004, a novel courmarin derivative, protects against oxygen-and glucose-deprivation/restoration-induced apoptosis in PC12 cells. Eur J Pharmacol 723:259–266

4. Zuo W, Chen J, Zhang S, Tang J, Liu H et al (2014) IMM-H004 prevents toxicity induced by delayed treatment of tPA in a rat model of focal cerebral ischemia involving PKA-and PI3K-dependent Akt activation. Eur J Neurosci 39:2107–2118

5. Zuo W, Zhang W, Han N, Chen NH (2015) Compound IMM-H004, a novel coumarin derivative, protects against CA1 cell loss and spatial learning impairments resulting from transient global ischemia. CNS Neurosci Ther 21:280–288

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