Enhanced cleavage of APP by co-expressed Bace1 alters the distribution of APP and its fragments in neuronal and non-neuronal cells-Reference-Cited by-同舟云学术

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Enhanced cleavage of APP by co-expressed Bace1 alters the distribution of APP and its fragments in neuronal and non-neuronal cells

Published:2022-03-09 Issue:5 Volume:59 Page:3073-3090
ISSN:0893-7648
Container-title:Molecular Neurobiology
language:en
Short-container-title:Mol Neurobiol

Author:

Aow Jonathan,Huang Tzu-Rung,Thinakaran Gopal,Koo Edward H.

Funder

national institutes of health

national research foundation singapore

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology

Link

https://link.springer.com/content/pdf/10.1007/s12035-022-02733-6.pdf

Reference44 articles.

1. Selkoe DJ, Hardy J (2016) The amyloid hypothesis of Alzheimer’s disease at 25 years. EMBO Mol Med 8(6):595–608

2. Nhan HS, Chiang K, Koo EH (2015) The multifaceted nature of amyloid precursor protein and its proteolytic fragments: friends and foes. Acta Neuropathol 129(1):1–19

3. Haass C, Kaether C, Thinakaran G, Sisodia S (2012) Trafficking and proteolytic processing of APP. Cold Spring Harb Perspect Med. 2(5):a006270

4. Rajendran L, Annaert W (2012) Membrane trafficking pathways in Alzheimer’s disease. Traffic 13(6):759–770

5. Toh WH, Gleeson PA (2016) Dysregulation of intracellular trafficking and endosomal sorting in Alzheimer’s disease: controversies and unanswered questions. Biochem J 473(14):1977–1993

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3. Evidence for a clathrin-independent endocytic pathway for APP internalization in the neuronal somatodendritic compartment;Cell Reports;2023-07

4. APP-BACE1 Interaction and Intracellular Localization Regulate Aβ Production in iPSC-Derived Cortical Neurons;Cellular and Molecular Neurobiology;2023-06-24

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