Increases of Galectin-1 and its S-nitrosylated form in the Brain Tissues of Scrapie-Infected Rodent Models and Human Prion Diseases-Reference-Cited by-同舟云学术

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Increases of Galectin-1 and its S-nitrosylated form in the Brain Tissues of Scrapie-Infected Rodent Models and Human Prion Diseases

Published:2016-05-23 Issue:5 Volume:54 Page:3707-3716
ISSN:0893-7648
Container-title:Molecular Neurobiology
language:en
Short-container-title:Mol Neurobiol

Author:

Guo Yan-Jun,Shi Qi,Yang Xiao-Dong,Li Jian-Le,Ma Yue,Xiao Kang,Chen Cao,Han Jun,Dong Xiao-Ping

Funder

Chinese National Natural Science Foundation Grant

China Mega-Project for Infectious Disease

SKLID Development Grant

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology

Link

http://link.springer.com/article/10.1007/s12035-016-9923-1/fulltext.html

Reference41 articles.

1. Liberski PP (2012) Historical overview of prion diseases: a view from afar. Folia Neuropathol 50:1–12

2. DeArmond SJ (2004) Discovering the mechanisms of neurodegeneration in prion diseases. Neurochem Res 29:1979–1998

3. Lv Y, Chen C, Zhang BY, Xiao K, Wang J et al (2015) Remarkable activation of the complement system and aberrant neuronal localization of the membrane attack complex in the brain tissues of scrapie-infected rodents. Mol Neurobiol 52:1165–1179

4. Prusiner SB (1998) The prion diseases. Brain Pathol 8:499–513

5. Colby DW, Prusiner SB (2011) Prions. Cold Spring Harb Perspect Biol 3:a006833

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Activation of Innate Immunity and Autophagy in Brain Tissues with Prion Disease and Degradation of Abnormal PrPs in Cells - China's Studies;CHINA CDC WEEKLY;2022

2. Association between Galectin Levels and Neurodegenerative Diseases: Systematic Review and Meta-Analysis;Biomolecules;2022-07-31

3. Galectins and Their Ligand Glycoconjugates in the Central Nervous System Under Physiological and Pathological Conditions;Frontiers in Neuroanatomy;2021-10-15

4. Significant enhanced expressions of aquaporin-1, -4 and -9 in the brains of various prion diseases;Prion;2019-01-01

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