Laforin Prevents Stress-Induced Polyglucosan Body Formation and Lafora Disease Progression in Neurons-Reference-Cited by-同舟云学术

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Laforin Prevents Stress-Induced Polyglucosan Body Formation and Lafora Disease Progression in Neurons

Published:2013-04-02 Issue:1 Volume:48 Page:49-61
ISSN:0893-7648
Container-title:Molecular Neurobiology
language:en
Short-container-title:Mol Neurobiol

Author:

Wang Yin,Ma Keli,Wang Peixiang,Baba Otto,Zhang Helen,Parent Jack M.,Zheng Pan,Liu Yang,Minassian Berge A.,Liu Yan

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology

Link

http://link.springer.com/content/pdf/10.1007/s12035-013-8438-2.pdf

Reference46 articles.

1. Cavanagh JB (1999) Corpora-amylacea and the family of polyglucosan diseases. Brain Res Brain Res Rev 29:265–295

2. Gambetti P, Di Mauro S, Hirt L, Blume RP (1971) Myoclonic epilepsy with lafora bodies. Some ultrastructural, histochemical, and biochemical aspects. Arch Neurol 25:483–493

3. Minassian BA (2002) Progressive myoclonus epilepsy with polyglucosan bodies: Lafora disease. Adv Neurol 89:199–210

4. Sakai M, Austin J, Witmer F, Trueb L (1970) Studies in myoclonus epilepsy (Lafora body form). II. Polyglucosans in the systemic deposits of myoclonus epilepsy and in corpora amylacea. Neurology 20:160–176

5. Chan EM, Young EJ, Ianzano L, Munteanu I, Zhao X, Christopoulos CC, Avanzini G, Elia M, Ackerley CA, Jovic NJ, Bohlega S, Andermann E, Rouleau GA, Delgado-Escueta AV, Minassian BA, Scherer SW (2003) Mutations in NHLRC1 cause progressive myoclonus epilepsy. Nat Genet 35:125–127

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