DNA Methylation—a Potential Source of Mitochondria DNA Base Mismatch in the Development of Diabetic Retinopathy-Reference-Cited by-同舟云学术

DNA Methylation—a Potential Source of Mitochondria DNA Base Mismatch in the Development of Diabetic Retinopathy

Published:2018-04-21 Issue:1 Volume:56 Page:88-101
ISSN:0893-7648
Container-title:Molecular Neurobiology
language:en
Short-container-title:Mol Neurobiol

Author:

Mishra Manish,Kowluru Renu A.^ORCID

Funder

National Eye Institute

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology

Link

http://link.springer.com/article/10.1007/s12035-018-1086-9/fulltext.html

Reference51 articles.

1. Brownlee M (2001) Biochemistry and molecular cell biology of diabetic complications. Nature 414:813–820. https://doi.org/10.1038/414813a

2. Frank RN (2004) Diabetic retinopathy. N Engl J Med 350:48–58. https://doi.org/10.1056/NEJMra1005073

3. Kern TS, Tang J, Mizutani M, Kowluru R, Nagraj R, Lorenzi M (2000) Response of capillary cell death to aminoguanidine predicts the development of retinopathy: comparison of diabetes and galactosemia. Invest Ophthalmol Vis Sci 41:3972–3978

4. Kowluru RA (2013) Mitochondria damage in the pathogenesis of diabetic retinopathy and in the metabolic memory associated with its continued progression. Curr Med Chem 20:3226–3233

5. Kowluru RA, Chan PS (2010) Metabolic memory in diabetes—from in vitro oddity to in vivo problem: role of apoptosis. Brain Res Bull 81:297–302. https://doi.org/10.1016/j.brainresbull.2009.05.006

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