Modulators of Neuroinflammation Have a Beneficial Effect in a Lafora Disease Mouse Model
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology
Link
https://link.springer.com/content/pdf/10.1007/s12035-021-02285-1.pdf
Reference65 articles.
1. Turnbull J, Tiberia E, Striano P, Genton P, Carpenter S, Ackerley CA et al (2016) Lafora disease. Epileptic Disord 18(S2):38–62
2. Sakai M, Austin J, Witmer F, Trueb L (1970) Studies in myoclonus epilepsy (Lafora body form). II. Polyglucosans in the systemic deposits of myoclonus epilepsy and in corpora amylacea. Neurology 20(2):160–176
3. Lafora GR, Glueck B (1911) Beitrag zur histogpathologie der myoklonischen epilepsie. Gesamte Neurol Psychiatr 6:1–14
4. Minassian BA, Lee Jeffrey R, Herbrick JA, Huizenga J, Soder S, Mungall AJ et al (1998) Mutations in a gene encoding a novel protein tyrosine phosphatase cause progressive myoclonus epilepsy. Nat Genet 20(2):171–174
5. Serratosa JM, Gómez-Garre P, Gallardo ME, Anta B (1999) Beltrán-Valero De Bernabé D, Lindhout D, et al. A novel protein tyrosine phosphatase gene is mutated in progressive myoclonus epilepsy of the Lafora type (EPM2). Hum Mol Genet 8(2):345–352
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