Mitochondrial Quality and Quantity Control: Mitophagy Is a Potential Therapeutic Target for Ischemic Stroke
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology
Link
https://link.springer.com/content/pdf/10.1007/s12035-022-02795-6.pdf
Reference145 articles.
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2. Deb P, Sharma S, Hassan KM (2010) Pathophysiologic mechanisms of acute ischemic stroke: an overview with emphasis on therapeutic significance beyond thrombolysis. Pathophysiology 17(3):197–218. https://doi.org/10.1016/j.pathophys.2009.12.001
3. Adibhatla RM, Hatcher JF (2008) Tissue plasminogen activator (tPA) and matrix metalloproteinases in the pathogenesis of stroke: therapeutic strategies. CNS Neurol Disord Drug Targets 7(3):243–253. https://doi.org/10.2174/187152708784936608
4. Henninger N, Fisher M (2016) Extending the time window for endovascular and pharmacological reperfusion. Transl Stroke Res 7(4):284–293. https://doi.org/10.1007/s12975-015-0444-4
5. Aronowski J, Strong R, Grotta JC (1997) Reperfusion injury: demonstration of brain damage produced by reperfusion after transient focal ischemia in rats. J Cereb Blood Flow Metab 17(10):1048–1056. https://doi.org/10.1097/00004647-199710000-00006
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