The Putative Drosophila TMEM184B Ortholog Tmep Ensures Proper Locomotion by Restraining Ectopic Firing at the Neuromuscular Junction
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology
Link
https://link.springer.com/content/pdf/10.1007/s12035-022-02760-3.pdf
Reference62 articles.
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3. Brusich DJ, Spring AM, James TD et al (2018) Drosophila CaV2 channels harboring human migraine mutations cause synapse hyperexcitability that can be suppressed by inhibition of a Ca2+store release pathway. PLoS Genet 14. https://doi.org/10.1371/journal.pgen.1007577
4. Stern M, Ganetzky B (1989) Altered synaptic transmission in drosophila hyperkinetic mutants. J Neurogenet 5:215–228. https://doi.org/10.3109/01677068909066209
5. Uytterhoeven V, Kuenen S, Kasprowicz J et al (2011) Loss of Skywalker reveals synaptic endosomes as sorting stations for synaptic vesicle proteins. Cell 145:117–132. https://doi.org/10.1016/j.cell.2011.02.039
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1. Pathogenic variants inTMEM184Bcause a neurodevelopmental syndrome via alteration of metabolic signaling;2024-07-01
2. Transmembrane protein 184B (TMEM184B) promotes expression of synaptic gene networks in the mouse hippocampus;BMC Genomics;2023-09-20
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