Downregulated Glia Interplay and Increased miRNA-155 as Promising Markers to Track ALS at an Early Stage-Reference-Cited by-同舟云学术

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Downregulated Glia Interplay and Increased miRNA-155 as Promising Markers to Track ALS at an Early Stage

Published:2017-06-13 Issue: Volume: Page:
ISSN:0893-7648
Container-title:Molecular Neurobiology
language:en
Short-container-title:Mol Neurobiol

Author:

Cunha Carolina,Santos Catarina,Gomes Cátia,Fernandes Adelaide,Correia Alexandra Marçal,Sebastião Ana Maria,Vaz Ana Rita,Brites Dora^ORCID

Funder

Fundação para a Ciência e a Tecnologia

Research Grant of the Santa Casa Scientific Research Program on ALS

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology

Link

http://link.springer.com/content/pdf/10.1007/s12035-017-0631-2.pdf

Reference75 articles.

1. Benatar M, Wuu J (2012) Presymptomatic studies in ALS: rationale, challenges, and approach. Neurology 79(16):1732–1739. doi: 10.1212/WNL.0b013e31826e9b1d

2. Rocha MC, Pousinha PA, Correia AM, Sebastiao AM, Ribeiro JA (2013) Early changes of neuromuscular transmission in the SOD1(G93A) mice model of ALS start long before motor symptoms onset. PLoS One 8(9):e73846. doi: 10.1371/journal.pone.0073846

3. Nascimento F, Pousinha PA, Correia AM, Gomes R, Sebastiao AM, Ribeiro JA (2014) Adenosine A2A receptors activation facilitates neuromuscular transmission in the pre-symptomatic phase of the SOD1(G93A) ALS mice, but not in the symptomatic phase. PLoS One 9(8):e104081. doi: 10.1371/journal.pone.0104081

4. Nascimento F, Sebastiao AM, Ribeiro JA (2015) Presymptomatic and symptomatic ALS SOD1(G93A) mice differ in adenosine A1 and A2A receptor-mediated tonic modulation of neuromuscular transmission. Purinergic Signal 11(4):471–480. doi: 10.1007/s11302-015-9465-4

5. Gravel M, Beland LC, Soucy G, Abdelhamid E, Rahimian R, Gravel C, Kriz J (2016) IL-10 controls early microglial phenotypes and disease onset in ALS caused by misfolded superoxide dismutase 1. J Neurosci 36(3):1031–1048. doi: 10.1523/JNEUROSCI.0854-15.2016

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