Role of constitutive nitric oxide synthase S-nitrosylation in Helicobacter pylori-induced gastric mucosal cell apoptosis: effect of ghrelin
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology (medical),Pharmacology,Immunology
Link
http://link.springer.com/content/pdf/10.1007/s10787-010-0051-7.pdf
Reference35 articles.
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3. Cuzzocrea S, Salvemini D (2007) Molecular mechanisms involved in the reciprocal regulation of cyclooxygenase and nitric oxide synthase enzymes. Kidney Int 71:290–297
4. de Boer WA (2000) Topics in Helicobacter pylori infection: focus on a ‘search-and-treat’ strategy for ulcer disease. Scand J Gastroenterol 35(suppl 32):4–9
5. Erwin PA, Lin AJ, Golan E et al (2005) Receptor-regulated dynamic S-nitrosylation of endothelial nitric-oxide synthase in vascular endothelial cells. J Biol Chem 280:19888–19894
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2. Role of LPS-elicited signaling in triggering gastric mucosal inflammatory responses to H. pylori: modulatory effect of ghrelin;Inflammopharmacology;2017-05-17
3. Ghrelin protects against palmitic acid or lipopolysaccharide-induced hepatocyte apoptosis through inhibition of MAPKs/iNOS and restoration of Akt/eNOS pathways;Biomedicine & Pharmacotherapy;2016-12
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