Baricitinib relieves DSS-induced ulcerative colitis in mice by suppressing the NF-κB and JAK2/STAT3 signalling pathways-Reference-Cited by-同舟云学术

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Baricitinib relieves DSS-induced ulcerative colitis in mice by suppressing the NF-κB and JAK2/STAT3 signalling pathways

Published:2024-01-16 Issue:1 Volume:32 Page:849-861
ISSN:0925-4692
Container-title:Inflammopharmacology
language:en
Short-container-title:Inflammopharmacol

Author:

Wu Qiong,Liu Yangyang,Liang Jinmeiqi,Dai Ao,Du Boyu,Xi Xueyan,Jin Lan,Guo Yang^ORCID

Funder

Key Technology Research and Development Program of Shandong

the Biomedical Research Foundation, Hubei University of Medicine

the Open Project of Hubei Key Laboratory of Wudang Local Chinese Medicine Research

Publisher

Springer Science and Business Media LLC

Link

https://link.springer.com/content/pdf/10.1007/s10787-023-01396-6.pdf

Reference29 articles.

1. Allocca M, Jovani M, Fiorino G et al (2013) Anti-IL-6 treatment for inflammatory bowel diseases: next cytokine, next target. Curr Drug Targets 14(12):1508–1521. https://doi.org/10.2174/13894501113146660224

2. Ashwood P, Harvey R, Verjee T et al (2004) Functional interactions between mucosal IL-1, IL-ra and TGF-beta 1 in ulcerative colitis. Inflamm Res 53(2):53–59. https://doi.org/10.1007/s00011-003-1219-z

3. Atreya R, Neurath MF (2005) Involvement of IL-6 in the pathogenesis of inflammatory bowel disease and colon cancer. Clin Rev Allergy Immunol 28(3):187–196. https://doi.org/10.1385/CRIAI:28:3:187

4. Barbulescu A, Askling J, Chatzidionysiou K et al (2022) Effectiveness of baricitinib and tofacitinib compared with bDMARDs in RA: results from a cohort study using nationwide Swedish register data. Rheumatology (oxford) 61(10):3952–3962. https://doi.org/10.1093/rheumatology/keac068

5. Barnes PJ, Karin M (1997) Nuclear factor-kappaB: a pivotal transcription factor in chronic inflammatory diseases. N Engl J Med 336(15):1066–1071. https://doi.org/10.1056/NEJM199704103361506

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