Increased neurovirulence and reactivation of the herpes simplex virus type 1 latency-associated transcript (LAT)-negative mutant dLAT2903 with a disrupted LAT miR-H2-Reference-Cited by-同舟云学术

Increased neurovirulence and reactivation of the herpes simplex virus type 1 latency-associated transcript (LAT)-negative mutant dLAT2903 with a disrupted LAT miR-H2

Published:2015-06-12 Issue:1 Volume:22 Page:38-49
ISSN:1355-0284
Container-title:Journal of NeuroVirology
language:en
Short-container-title:J. Neurovirol.

Author:

Jiang Xianzhi,Brown Don,Osorio Nelson,Hsiang Chinhui,BenMohamed Lbachir,Wechsler Steven L.

Publisher

Springer Science and Business Media LLC

Subject

Virology,Cellular and Molecular Neuroscience,Clinical Neurology,Neurology

Link

http://link.springer.com/content/pdf/10.1007/s13365-015-0362-y.pdf

Reference53 articles.

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3. Allen SJ, Rhode-Kurnow A, Mott KR, Jiang X, Carpenter D, Rodriguez-Barbosa JI, Jones C, Wechsler SL, Ware CF, Ghiasi H (2014) Interactions between herpesvirus entry mediator (TNFRSF14) and latency-associated transcript during herpes simplex virus 1 latency. J Virol 88:1961–1971, PMC:3911542

4. Block TM, Deshmane S, Masonis J, Maggioncalda J, Valyi-Nagi T, Fraser NW (1993) An HSV LAT null mutant reactivates slowly from latent infection and makes small plaques on CV-1 monolayers. Virology 192:618–630

5. Bloom DC, Devi-Rao GB, Hill JM, Stevens JG, Wagner EK (1994) Molecular analysis of herpes simplex virus type 1 during epinephrine-induced reactivation of latently infected rabbits in vivo. J Virol 68:1283–1292

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