JC polyomavirus expression and bell-shaped regulation of its SF2/ASF suppressor during the follow-up of multiple sclerosis patients treated with natalizumab
Author:
Funder
CARITRO
Regione Sardegna
Publisher
Springer Science and Business Media LLC
Subject
Virology,Cellular and Molecular Neuroscience,Clinical Neurology,Neurology
Link
http://link.springer.com/content/pdf/10.1007/s13365-016-0492-x.pdf
Reference62 articles.
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2. Atwood WJ, Amemiya K, Traub R, Harms J, Major EO (1992) Interaction of the human polyomavirus, JCV, with human B-lymphocytes. Virology 190:716–723
3. Baroncini D, Ghezzi A, Annovazzi PO, Colombo B, Martinelli V, Minonzio G, Moiola L, Rodegher M, Zaffaroni M, Comi G (2016) Natalizumab versus fingolimod in patients with relapsing-remitting multiple sclerosis non-responding to first-line injectable therapies. Mult Scler 22:1315–1326
4. Berger JR, Fox RJ (2016) Reassessing the risk of natalizumab-associated PML. J Neurovirol 22:533–535
5. Bloomgren G, Richman S, Hotermans C, Subramanyan M, Goelz S, Natarajan A, Lee S, Plavina T, Scanlon JV, Sandrock A, Bozic C (2012) Risk of Natalizumab-associated progressive multifocal leukoencephalopathy. N Engl J Med 366:1870–1880
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2. Early reduction of the splicing factor2/alternative splicing factor: a cellular inhibitor of the JC polyomavirus in natalizumab-treated MS patients long before developing progressive multifocal leukoencephalopathy;Journal of NeuroVirology;2019-08-29
3. PML with dimethyl fumarate—No role for natalizumab?;Multiple Sclerosis Journal;2019-08-22
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5. Understanding Progressive Multifocal Leukoencephalopathy Risk in Multiple Sclerosis Patients Treated with Immunomodulatory Therapies: A Bird’s Eye View;Frontiers in Immunology;2018-02-02
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