Pseudohyphal differentiation defect due to mutations in GPCR and ammonium signaling is suppressed by low glucose concentration: a possible integrated role for carbon and nitrogen limitation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00294-008-0202-1.pdf
Reference46 articles.
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2. Adams A, Gottschling DE, Kaiser CA, Stearns T (1997) Methods in yeast genetics. Cold Spring Harbor Laboratory, New York, pp 1–157
3. Beck T, Hall MN (1999) The TOR signaling pathway controls nuclear localization of nutrient-regulated transcription factors. Nature 402:689–692
4. Betram PG, Choi JH, Carvalho J, Chan T, Ai W, Steven Zheng XF (2002) Convergence of TOR-nitrogen and Snf1-glucose signaling pathways onto Gln3. Mol Cell Biol 22:1246–1252
5. Boeckstaens M, Andre B, Marini AM (2007) The yeast ammonium transport protein Mep2 and its positive regulator, the Npr1 kinase, play an important role in normal and pseudohyphal growth on various nitrogen media through retrieval of excreted ammonium. Mol Microbiol 64:534–546
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