Growing Spectrum of Autoimmune Nodopathies
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neurology (clinical),General Neuroscience
Link
https://link.springer.com/content/pdf/10.1007/s11910-023-01264-4.pdf
Reference75 articles.
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2. Querol L, Nogales-Gadea G, Rojas-Garcia R, Martinez-Hernandez E, Diaz-Manera J, Suárez-Calvet X, Navas M, Araque J, Gallardo E, Illa I. Antibodies to contactin-1 in chronic inflammatory demyelinating polyneuropathy. Ann Neurol. 2013;73(3):370–80. https://doi.org/10.1002/ana.23794.
3. Ng JK, Malotka J, Kawakami N, Derfuss T, Khademi M, Olsson T, Linington C, Odaka M, Tackenberg B, Prüss H, et al. Neurofascin as a target for autoantibodies in peripheral neuropathies. Neurology. 2012;79(23):2241–8. https://doi.org/10.1212/WNL.0b013e31827689ad.
4. Querol L, Nogales-Gadea G, Rojas-Garcia R, Diaz-Manera J, Pardo J, Ortega-Moreno A, Sedano MJ, Gallardo E, Berciano J, Blesa R, et al. Neurofascin IgG4 antibodies in CIDP associate with disabling tremor and poor response to IVIg. Neurology. 2014;82(10):879–86. https://doi.org/10.1212/wnl.0000000000000205.
5. Manso C, Querol L, Lleixà C, Poncelet M, Mekaouche M, Vallat JM, Illa I, Devaux JJ. Anti-neurofascin-155 IgG4 antibodies prevent paranodal complex formation in vivo. J Clin Invest. 2019;129(6):2222–36. https://doi.org/10.1172/jci124694. In this paper, authors demonstrated the pathogenic mechanisms of NF155 IgG4 antibodies. These antibodies bind to NF155 on the schwann cells and delpete its level resulting in paranodal dysfunction.
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