Psoriasis: from Pathogenesis to Targeted Therapies
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine,Immunology and Allergy
Link
http://link.springer.com/article/10.1007/s12016-018-8668-1/fulltext.html
Reference80 articles.
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2. Tonel G, Conrad C (2009) Interplay between keratinocytes and immune cells—recent insights into psoriasis pathogenesis. Int J Biochem Cell Biol 41(5):963–968. https://doi.org/10.1016/j.biocel.2008.10.022
3. Aggarwal S, Ghilardi N, Xie MH, de Sauvage FJ, Gurney AL (2003) Interleukin-23 promotes a distinct CD4 T cell activation state characterized by the production of interleukin-17. J Biol Chem 278(3):1910–1914. https://doi.org/10.1074/jbc.M207577200
4. Hijnen D, Knol EF, Gent YY, Giovannone B, Beijn SJ, Kupper TS, Bruijnzeel-Koomen CA, Clark RA (2013) CD8(+) T cells in the lesional skin of atopic dermatitis and psoriasis patients are an important source of IFN-gamma, IL-13, IL-17, and IL-22. J Invest Dermatol 133(4):973–979. https://doi.org/10.1038/jid.2012.456
5. Di Meglio P, Villanova F, Navarini AA, Mylonas A, Tosi I, Nestle FO, Conrad C (2016) Targeting CD8(+) T cells prevents psoriasis development. J Allergy Clin Immunol 138(1):274–276 e276. https://doi.org/10.1016/j.jaci.2015.10.046
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