Arsenic trioxide alleviates airway hyperresponsiveness and promotes apoptosis of CD4+ T lymphocytes: evidence for involvement of the ER stress–CHOP pathway

Author:

Li K.,Zhang L.,Xiang X.,Gong S.,Ma L.,Xu L.,Wang G.,Liu Y.,Ji X.,Liu S.,Chen P.,Zeng H.,Li J.

Publisher

Springer Science and Business Media LLC

Subject

General Medicine

Reference28 articles.

1. Arm JP, Lee TH (1992) The pathobiology of bronchial asthma. Adv Immunol 51:323–382

2. Qin DY, Huang R, Wu T (2008) In vitro arsenic trioxide induces apoptosis in T cells of asthmatic patients by a Bcl-2 related mechanism. Yao Xue Xue Bao 43(1):35–43

3. Chen C-J, Chen CW, Wu M-M, Kuo T-L (1992) Cancer potential in liver, lung, bladder and kidney due to ingested inorganic arsenic in drinking water. Br J Cancer 66:888–892

4. Zhou LF, Yin KS (2002) Effect of arsenic trioxide on apoptosis of pulmonary eosinophils in asthmatic guinea-pigs. Zhongguo Zhong Xi Yi Jie He Za Zhi 22:292–294

5. Chu KH, Lee CC, Hsin SC, Cai BC, Wang JH, Chiang BL (2010) Arsenic trioxide alleviates airway hyperresponsiveness and eosinophilia in a murine model of asthma. Cell Mol Immunol 5:375–380

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