Targeted treatments of AL and ATTR amyloidosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10741-021-10180-z.pdf
Reference124 articles.
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2. Manral P, Reixach N (2015) Amyloidogenic and non-amyloidogenic transthyretin variants interact differently with human cardiomyocytes: insights into early events of non-fibrillar tissue damage. Biosci Rep 35:e00172. https://doi.org/10.1042/BSR20140155
3. Merlini G, Seldin DC, Gertz MA (2011) Amyloidosis: pathogenesis and new therapeutic options. J Clin Oncol 29:1924–1933. https://doi.org/10.1200/JCO.2010.32.2271
4. Foss TR, Wiseman RL, Kelly JW (2005) The pathway by which the tetrameric protein transthyretin dissociates. Biochemistry 44:15525–15533. https://doi.org/10.1021/bi051608t
5. Kanda Y, Goodman DS, Canfield RE, Morgan FJ (1974) The amino acid sequence of human plasma prealbumin. J Biol Chem 249:6796–6805
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1. Subtyping of cardiac amyloidosis by mass spectrometry-based proteomics of endomyocardial biopsies;Amyloid;2022-10-09
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