UBTF mediates activation of L3MBTL2 to suppress NISCH expression through histone H2AK119 monoubiquitination modification in breast cancer
Author:
Funder
Dazhou Science and Technology Bureau Project
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s10585-024-10299-x.pdf
Reference41 articles.
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2. Hashemi M, Arani HZ, Orouei S, Fallah S, Ghorbani A, Khaledabadi M, Kakavand A, Tavakolpournegari A, Saebfar H, Heidari H, Salimimoghadam S, Entezari M, Taheriazam A, Hushmandi K (2022) EMT mechanism in breast cancer metastasis and drug resistance: revisiting molecular interactions and biological functions. Biomed Pharmacother 155:113774. https://doi.org/10.1016/j.biopha.2022.113774
3. Luond F, Sugiyama N, Bill R, Bornes L, Hager C, Tang F, Santacroce N, Beisel C, Ivanek R, Burglin T, Tiede S, van Rheenen J, Christofori G (2021) Distinct contributions of partial and full EMT to breast cancer malignancy. Dev Cell 56(23):3203–3221e3211. https://doi.org/10.1016/j.devcel.2021.11.006
4. Okpechi SC, Yousefi H, Nguyen K, Cheng T, Alahari NV, Collins-Burow B, Burow ME, Alahari SK (2022) Role of Nischarin in the pathology of diseases: a special emphasis on breast cancer. Oncogene 41(8):1079–1086. https://doi.org/10.1038/s41388-021-02150-4
5. Maziveyi M, Alahari SK (2015) Breast Cancer Tumor suppressors: a special emphasis on Novel protein nischarin. Cancer Res 75(20):4252–4259. https://doi.org/10.1158/0008-5472.CAN-15-1395
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