Expression of a phosphorylated form of ATF4 in lung and non-small cell lung cancer tissues
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s13277-013-1104-5.pdf
Reference19 articles.
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2. Ameri K, Lewis CE, Raida M, et al. Anoxic induction of ATF-4 through HIF-1-independent pathways of protein stabilization in human cancer cells. Blood. 2004;103(5):1876–82.
3. Oskolkova OV, Afonyushkin T, Leitner A, et al. ATF4-dependent transcription is a key mechanism in VEGF upregulation by oxidized phospholipids: critical role of oxidized sn-2 residues in activation of unfolded protein response. Blood. 2008;112(2):330–9.
4. Gjymishka A, Palii SS, Shan J, et al. Despite increased ATF4 Binding at the C/EBP-ATF composite site following activation of the unfolded protein response, system A transporter 2 (SNAT2) transcription activity is repressed in HepG2 cells. J Biol Chem. 2008;283(41):27736–47.
5. Xuan B, Qian Z, Torigoi E, et al. Human cytomegalovirus protein pUL38 induces ATF4 expression, inhibits persistent JNK phosphorylation, and suppresses endoplasmic reticulum stress-induced cell death. J Virol. 2009;83(8):3463–74.
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