Improving cardiac gap junction communication as a new antiarrhythmic mechanism: the action of antiarrhythmic peptides-Reference-Cited by-同舟云学术

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Improving cardiac gap junction communication as a new antiarrhythmic mechanism: the action of antiarrhythmic peptides

Published:2009-11-27 Issue:3 Volume:381 Page:221-234
ISSN:0028-1298
Container-title:Naunyn-Schmiedeberg's Archives of Pharmacology
language:en
Short-container-title:Naunyn-Schmied Arch Pharmacol

Author:

Dhein Stefan,Hagen Anja,Jozwiak Joanna,Dietze Anna,Garbade Jens,Barten Markus,Kostelka Martin,Mohr Friedrich-Wilhelm

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00210-009-0473-1.pdf

Reference104 articles.

1. Aonuma S, Kohama Y, Akai K, Komiyama Y, Nakajima S, Wakabayashi M et al (1980) Studies on heart. XIX. Isolation of an atrial peptide that improves the rhythmicity of cultured myocardial cell clusters. Chem Pharm Bull 28:3332–3339

2. Axelsen LN, Stahlhut M, Mohammed S, Larsen BD, Nielsen MS, Holstein-Rathlou NH, Andersen S, Jensen ON, Hennan JK, Kjølbye AL (2006) Identification of ischemia-regulated phosphorylation sites in connexin43: a possible target for the antiarrhythmic peptide analogue rotigaptide (ZP123). J Mol Cell Cardiol 40:790–798

3. Bedner P, Niessen H, Odermatt B, Willecke K, Harz H (2003) A method to determine the relative cAMP permeability of connexin channels. Exp Cell Res 291:25–35

4. Boldt A, Wetzel U, Lauschke J, Weigl J, Gummert J, Hindricks G, Kottkamp H, Dhein S (2004) Fibrosis in left atrial tissue of patients with atrial fibrillation with and without underlying mitral valve disease. Heart 90:400–405

5. Buchanan JW, Saito T, Gettes LS (1985) The effect of antiarrhythmic drugs, stimulation frequency and potassium induced resting membrane potential changes on conduction velocity and dV/dtmax in guinea pig myocardium. Circ Res 56:696–703

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