Increased nitric oxide activity compensates for increased oxidative stress to maintain endothelial function in rat aorta in early type 1 diabetes
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00210-012-0794-3.pdf
Reference66 articles.
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2. Adak S, Wang Q, Stuehr DJ (2000) Arginine conversion to nitroxide by tetrahydrobiopterin-free neuronal nitric-oxide synthase. Implications for mechanism. J Biol Chem 275:33554–33561
3. Alencar JL, Chalupsky K, Sarr M, Schini-Kerth V, Vanin AF, Stoclet J-C, Muller B (2003) Inhibition of arterial contraction by dinitrosyl-iron complexes: critical role of the thiol ligand in determining rate of nitric oxide (NO) release and formation of releasable NO stores by S-nitrosation. Biochem Pharmacol 66:2365–2374
4. Alper G, Olukman M, Irer S, Çalayan O, Duman E, Ylmaz C, Ülker S (2006) Effect of vitamin E and C supplementation combined with oral antidiabetic therapy on the endothelial dysfunction in the neonatally streptozotocin injected diabetic rat. Diabetes Metab Res Rev 22:190–197
5. Altan VM, Karasu C, Özüari A (1989) The effects of type-1 and type-2 diabetes on endothelium-dependent relaxation in rat aorta. Pharmacol Biochem Behav 33:519–522
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