The role of BUD31 in clear cell renal cell carcinoma: prognostic significance, alternative splicing, and tumor immune environment
Author:
Funder
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s10238-024-01451-8.pdf
Reference47 articles.
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2. Wang Z, Wang S, Qin J, et al. Splicing factor BUD31 promotes ovarian cancer progression through sustaining the expression of anti-apoptotic BCL2L12. Nat Commun. 2022;13:6246.
3. Hsu CL, Liu JS, Wu PL, et al. Identification of a new androgen receptor (AR) co-regulator BUD31 and related peptides to suppress wild-type and mutated AR-mediated prostate cancer growth via peptide screening and X-ray structure analysis. Mol Oncol. 2014;8:1575–87.
4. Hsu TY, Simon LM, Neill NJ, et al. The spliceosome is a therapeutic vulnerability in MYC-driven cancer. Nature. 2015;525:384–8.
5. Sanjuan-Sanjuan A, Alors-Perez E, Sanchez-Frias M, et al. Splicing machinery is impaired in oral squamous cell carcinomas and linked to key pathophysiological features. Int J Mol Sci. 2024;25(13):2969.
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