Silencing of PCDH10 in hepatocellular carcinoma via de novo DNA methylation independent of HBV infection or HBX expression-Reference-Cited by-同舟云学术

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Silencing of PCDH10 in hepatocellular carcinoma via de novo DNA methylation independent of HBV infection or HBX expression

Published:2012-04-29 Issue:2 Volume:13 Page:127-134
ISSN:1591-8890
Container-title:Clinical and Experimental Medicine
language:en
Short-container-title:Clin Exp Med

Author:

Fang Song,Huang Shi-feng,Cao Ju,Wen Yang-an,Zhang Li-Ping,Ren Guo-Sheng

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s10238-012-0182-9.pdf

Reference21 articles.

1. Fung J, Lai CL, Yuen MF (2009) Hepatitis B and C virus-related carcinogenesis. Clin Microbiol Infect 15:964–970

2. Jicai Z, Zongtao Y, Jun L et al (2006) Persistent infection of hepatitis B virus is involved in high rate of p16 methylation in hepatocellular carcinoma. Mol Carcinog 45:530–536

3. Zondervan PE, Wink J, Alers JC (2000) Molecular cytogenetic evaluation of virus-associated and non-viral hepatocellular carcinoma: analysis of 26 carcinomas and 12 concurrent dysplasias. J Pathol 192:207–215

4. Neuveut C, Wei Y, Buendia MA (2010) Mechanisms of HBV-related hepatocarcinogenesis. J Hepatol 52:594–604

5. Gearhart TL, Bouchard MJ (2010) Replication of the hepatitis B virus requires a calcium-dependent HBx-induced G1 phase arrest of hepatocytes. Virology 407:14–25

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