Biomaterial-based immunotherapeutic strategies for rheumatoid arthritis
Author:
Funder
National Institute of Allergy and Infectious Diseases
National Institute of General Medical Sciences
Publisher
Springer Science and Business Media LLC
Subject
Pharmaceutical Science
Link
https://link.springer.com/content/pdf/10.1007/s13346-021-01038-w.pdf
Reference187 articles.
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2. McInnes IB, Schett G. The pathogenesis of rheumatoid arthritis. N Engl J Med. 2011;365(23):2205–19.
3. Deane KD, Demoruelle MK, Kelmenson LB, Kuhn KA, Norris JM, Holers VM. Genetic and environmental risk factors for rheumatoid arthritis. Best Pract Res Clin Rheumatol [Internet]. 2017;31(1):3–18. Available from: https://doi.org/10.1016/j.berh.2017.08.003
4. Coras R, Murillo-Saich J, Guma M. Circulating pro- and anti-inflammatory metabolites and its potential role in rheumatoid arthritis pathogenesis. Cells [Internet]. 2020 Mar 30 [cited 2021 Jun 20];9(4):827. Available from: https://www.mdpi.com/2073-4409/9/4/827
5. Holoshitz J. The rheumatoid arthritis HLA-DRB1 shared epitope [Internet]. Vol. 22, Current Opinion in Rheumatology. NIH Public Access; 2010 [cited 2020 Dec 23]. p. 293–8. Available from: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2921962/?report=abstract
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