Selective inhibition of oncogenic ras- p21 in vivo by agents that block its interaction with jun -N-kinase (JNK) and jun proteins. Implications for the design of selective chemotherapeutic agents-Reference-Cited by-同舟云学术

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Selective inhibition of oncogenic ras- p21 in vivo by agents that block its interaction with jun -N-kinase (JNK) and jun proteins. Implications for the design of selective chemotherapeutic agents

Published:1997-11-15 Issue:1 Volume:41 Page:79-85
ISSN:0344-5704
Container-title:Cancer Chemotherapy and Pharmacology
language:
Short-container-title:Cancer Chemotherapy and Pharmacology

Author:

Amar Shazia,Glozman Albert,Chung Denise,Adler Victor,Ronai Zeev,Friedman Fred K.,Robinson Richard,Brandt-Rauf Paul,Yamaizumi Z.,Pincus Matthew R.

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Cancer Research,Pharmacology,Toxicology,Oncology

Link

http://link.springer.com/content/pdf/10.1007/s002800050711.pdf

Cited by 24 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The potential roles of c-Jun N-terminal kinase (JNK) during the maturation and aging of oocytes produced by a marine protostome worm;Zygote;2017-10-16

2. JNK does not regulate meiotic progression in Xenopus oocytes: The strange case of pJNK and pERK;Developmental Biology;2016-08

3. Trans-membrane peptide therapy for malignant glioma by use of a peptide derived from the MDM2 binding site of p53;Journal of Neuro-Oncology;2012-04-18

4. Oncoproteins and Early Tumor Detection;Henry's Clinical Diagnosis and Management by Laboratory Methods;2011

5. Two peptides derived from ras-p21 induce either phenotypic reversion or tumor cell necrosis of ras-transformed human cancer cells;Cancer Chemotherapy and Pharmacology;2007-12-08

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