Isolated pontine infarction versus pontine plus infarction: prevalence, pathogenic mechanism, and outcomes
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neurology (clinical),Neurology
Link
https://link.springer.com/content/pdf/10.1007/s00415-022-11075-1.pdf
Reference22 articles.
1. Bassetti C, Bogousslavsky J, Barth A, Regli F (1996) Isolated infarcts of the pons. Neurology 46:165–175
2. Kumral E, Bayulkem G, Evyapan D (2002) Clinical spectrum of pontine infarction. Clinical-MRI correlations. J Neurol 249:1659–1670. https://doi.org/10.1007/s00415-002-0879-x
3. Erro ME, Gallego J, Herrera M, Bermejo B (2005) Isolated pontine infarcts: etiopathogenic mechanisms. Eur J Neurol 12:984–988. https://doi.org/10.1111/j.1468-1331.2005.01119.x
4. Oh S, Bang OY, Chung CS, Lee KH, Chang WH, Kim GM (2012) Topographic location of acute pontine infarction is associated with the development of progressive motor deficits. Stroke 43:708–713. https://doi.org/10.1161/STROKEAHA.111.632307
5. Kim JS, Cho KH, Kang DW, Kwon SU, Suh DC (2009) Basilar artery atherosclerotic disease is related to subacute lesion volume increase in pontine base infarction. Acta Neurol Scand 120:88–93. https://doi.org/10.1111/j.1600-0404.2008.01124.x
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1. Functional Outcomes in Conservatively vs Surgically Treated Cerebellar Infarcts;JAMA Neurology;2024-04-01
2. Morphological predictors of neurological deterioration in patients with acute isolated pontine infarct;Acta Neurologica Belgica;2023-03-19
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