Cerebral aneurysm in three pediatric patients with STAT1 gain-of-function mutations
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neurology (clinical),Neurology
Link
https://link.springer.com/content/pdf/10.1007/s00415-022-11131-w.pdf
Reference18 articles.
1. Jayaraman T et al (2008) TNF-alpha-mediated inflammation in cerebral aneurysms: a potential link to growth and rupture. Vasc Health Risk Manag 4(4):805–817
2. Chalouhi N, Hoh BL, Hasan D (2013) Review of cerebral aneurysm formation, growth, and rupture. Stroke 44(12):3613–3622
3. Toubiana J et al (2016) Heterozygous STAT1 gain-of-function mutations underlie an unexpectedly broad clinical phenotype. Blood 127(25):3154–3164
4. Huang J et al (2005) Intracranial aneurysms in the pediatric population: case series and literature review. Surg Neurol 63(5):424–432
5. Backes D et al (2016) Patient- and aneurysm-specific risk factors for intracranial aneurysm growth. Stroke 47(4):951–957
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1. Current understanding of macrophages in intracranial aneurysm: relevant etiological manifestations, signaling modulation and therapeutic strategies;Frontiers in Immunology;2024-01-08
2. Interferonopathies masquerading as non-Mendelian autoimmune diseases: pattern recognition for early diagnosis;Frontiers in Pediatrics;2023-08-09
3. Neutrophils in STAT1 Gain-Of-Function Have a Pro-inflammatory Signature Which Is Not Rescued by JAK Inhibition;Journal of Clinical Immunology;2023-06-26
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