KDM5B promotes cell migration by regulating the noncanonical Wnt/PCP pathway in Hirschsprung’s disease
Author:
Funder
the doctoral fund of the first affiliated hospital of harbin medical university
Publisher
Springer Science and Business Media LLC
Subject
General Medicine,Surgery,Pediatrics, Perinatology and Child Health
Link
https://link.springer.com/content/pdf/10.1007/s00383-021-05005-x.pdf
Reference23 articles.
1. Borrego S, Ruiz-Ferrer M, Fernandez RM, Antinolo G (2013) Hirschsprung's disease as a model of complex genetic etiology. Histol Histopathol 28:1117–1136. https://doi.org/10.14670/HH-28.1117
2. Amiel J, Sproat-Emison E, Garcia-Barcelo M et al (2008) Hirschsprung disease, associated syndromes and genetics: a review. J Med Genet 45:1–14. https://doi.org/10.1136/jmg.2007.053959
3. Jaroy EG, Acosta-Jimenez L, Hotta R, Goldstein AM, Emblem R, Klungland A, Ougland R (2019) Too much guts and not enough brains: (epi)genetic mechanisms and future therapies of Hirschsprung disease—a review. Clin Epigenetics 11:135. https://doi.org/10.1186/s13148-019-0718-x
4. Schmitz SU, Albert M, Malatesta M, Morey L, Johansen JV, Bak M, Tommerup N, Abarrategui I, Helin K (2011) Jarid1b targets genes regulating development and is involved in neural differentiation. EMBO J 30:4586–4600. https://doi.org/10.1038/emboj.2011.383
5. Hallaq R, Volpicelli F, Cuchillo-Ibanez I et al (2015) The Notch intracellular domain represses CRE-dependent transcription. Cell Signal 27:621–629. https://doi.org/10.1016/j.cellsig.2014.11.034
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