Portal vein thrombosis patients harboring JAK2V617F have poor long-term outcomes despite anticoagulation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine,Hematology
Link
http://link.springer.com/content/pdf/10.1007/s11239-020-02052-4.pdf
Reference19 articles.
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2. Dentali F, Squizzato A, Brivio L, Appio L, Campiotti L, Crowther M et al (2009) JAK2V617F mutation for the early diagnosis of Ph-myeloproliferative neoplasms in patients with venous thromboembolism: a meta-analysis. Blood 113(22):5617–5623
3. How J, Zhou A, Oh ST (2017) Splanchnic vein thrombosis in myeloproliferative neoplasms: pathophysiology and molecular mechanisms of disease. Ther Adv Hematol 8(3):107–118
4. Sozer S, Fiel MI, Schiano T, Xu M, Mascarenhas J, Hoffman R (2009) The presence of JAK2V617F mutation in the liver endothelial cells of patients with Budd-Chiari syndrome. Blood 113(21):5246–5249
5. Plessier A, Darwish-Murad S, Hernandez-Guerra M, Consigny Y, Fabris F, Trebicka J et al (2010) Acute portal vein thrombosis unrelated to cirrhosis: a prospective multicenter follow-up study. Hepatology 51(1):210–218
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2. Percutaneous mesocaval shunt creation for portal thrombosis in a patient with a JAK2V617F mutation;Thrombosis Research;2024-02
3. Decreased patency of transjugular intrahepatic portosystemic shunts performed for splanchnic vein thrombosis in patients with myeloproliferative neoplasms;Thrombosis Research;2024-02
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