High D dimers and low global fibrinolysis coexist in COVID19 patients: what is going on in there?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine,Hematology
Link
https://link.springer.com/content/pdf/10.1007/s11239-020-02226-0.pdf
Reference24 articles.
1. Fei Z et al (2020) Clinical course and risk factors for mortality of adult inpatients with COVID-19 in Wuhan, China: a retrospective cohort study. Lancet (London, England) 395(10229):1054–62 [cited 14 Jun 2020]. Available from https://pubmed.ncbi.nlm.nih.gov/32171076/?from_single_result=Lancet+Lond+Engl.+2020%3B395%2810229%29%3A
2. Wright FL et al (2020) Fibrinolysis shutdown correlates to thromboembolic events in severe COVID-19 infection. J Am Coll Surg [cited 14 Jun 2020]. Available from: https://pubmed.ncbi.nlm.nih.gov/32422349/
3. Schmitt FCF, Manolov V, Morgenstern J, Fleming T, Heitmeier S, Uhle F et al (2019) Acute fibrinolysis shutdown occurs early in septic shock and is associated with increased morbidity and mortality: results of an observational pilot study. Ann Intensive Care 9(1):19
4. Stettler GR, Moore EE, Moore HB, Nunns GR, Silliman CC, Banerjee A et al (2019) Redefining postinjury fibrinolysis phenotypes using two viscoelastic assays. J Trauma Acute Care Surg 86(4):679–685
5. Kuiper GJAJM, Kleinegris MCF, van Oerle R, Spronk HMH, Lancé MD, ten Cate H et al (2016) Validation of a modified thromboelastometry approach to detect changes in fibrinolytic activity. Thromb 14:1
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