Presence of CCK-B Receptor mRNA in Human Functionless Pituitary Tumors
Author:
Publisher
Kluwer Academic Publishers
Link
http://link.springer.com/content/pdf/10.1007/0-387-23761-5_50.pdf
Reference10 articles.
1. Mergery C Beinfeld, Wenger W (2002) CCK processing by pituitary GH3 cells, human teratocarcinoma cells NT2 and hNT differentiated human neuronal cells Evidence for a differentiation-induced change in enzyme expression and pro CCK processing. Life Sciences 70:1251–1258.
2. Smith AJ, Freedman SB (1996) CCK-B receptor-mediated stimulation of polyphosphoinositide turnover in GH3 pituitary cells in response to cholecystokinin and pentagastrin. Life Sciences 58:883–895.
3. Shinohara S, Kawasaki K (1994) Desensitization of cholecystokinin B receptors in GH3 cells. J Neurochemistry 62:1352–1356.
4. Kuwahara T, Takamiya M, Nagase H, et al (1993) GH3 cells, an anterior pituitary cell line, express CCK-B receptors. Peptides 14:647–649.
5. Smith AJ, McKernan RM (1999) Proliferative effects of cholecystokinin in GH3 pituitary cells mediated by CCK2 receptors and potentiated by insulin. Br J Pharmacology 126:79–86.
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