Synergistic inhibition of metabolic cooperation by oleic acid or 12-0-tetradecanoylphorbol-13-acetate and dichlorodiphenyltrichlorethane (DDT) in Chinese hamster V79 cells: Implication of a role for protein kinase C in the regulation of gap junctional intercellular communication

Author:

Aylsworth Charles F.,Trosko James E.,Chang Chia C.,Benjamin K.,Lockwood Elizabeth

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Cell Biology,Toxicology

Reference34 articles.

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2. AYLSWORTH, C. F., TROSKO, J.E. and WELSCH, C.W. (1986). Influence of lipids on gap junction-mediated intercellular communication between Chinese hamster cells in vitro. Cancer Res. 46:4527?4533.

3. BOUTWELL, R.K. (1984). The functions and mechanism of promoters of carcinogenesis. CRC Crit. Rev. Tox. 2:419?443.

4. CASTAGNA, M., TAKAI, Y., KAIBUCHI, K., SANO, K., KIKKAWA, U. and NISHIZUKA, Y. (1982). Direct activation of calcium-activated protein kinase by tumor-promoting phorbol esters. J. Biol. Chem. 257:7847?7851.

5. DAVIDSON, J.S., BAUMGARTEN, I. and HARLEY, E.H. (1985). Use of a new citrulline incorporation assay to investigate inhibition of intercellular communication by 1.1.1-trichloro-2.2-bis(p-chlorophenyl)-ethane in human fibroblasts. Cancer Res. 45:515?519.

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