The effect of the two epipodophyllotoxin derivatives etoposide (VP-16) and teniposide (VM-26) on cell lines established from patients with small cell carcinoma of the lung

Author:

Roed Henrik,Vindelov Lars L.,Christensen Ib J.,Spang-Thomsen Mogens,Hansen Heine Hoi

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Cancer Research,Pharmacology,Toxicology,Oncology

Reference22 articles.

1. Allen LM, Creaven PJ (1975) Comparison of the human pharmacokinetics of VM-26 and VP-16, two antineoplastic epipodophyllotoxin glucopyranoside derivatives. Eur J Cancer 11:697?707

2. Allen LM (1978) Comparison of uptake and binding of two epipodophyllotoxin glucopyranosides, 4?-demethyl epipodophyllotoxin thenylidene-?-D-glucoside ind 4?-demethyl epipodophyllotoxin ethylidene-?-D-glucoside in the L1210 leukemia cell. Cancer Res 38:2549?2554

3. Bork E, Hansen M, Dombernowsky P, Hansen SW, Pedersen AG, Hansen HH, (1986) Teniposide (VM-26), an overlooked highly active agent in small cell lung cancer. Results of a phase II trial. J Clin Oncol 4:524?527

4. Christensen I, Hartmann NR, Keiding N, Larsen JK, Noer H, Vindeløv L (1978) Statistical analysis of DNA distributions from cell populations with partial synchrony. In: Lutz D (ed) Pulsecytophotometry, part 3. European Press Medicon, Ghent, pp 71?78

5. Creaven PJ (1982) The clinical pharmacology of VM-26 and VP-16-213, a brief overview. Cancer Chemother Pharmacol 7: 133?140

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