Enigmatic PreS deletions in hepatitis B virus DNA
Author:
Funder
Justus-Liebig-Universität Gießen
Publisher
Springer Science and Business Media LLC
Subject
Virology,Genetics,Molecular Biology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11262-020-01805-w.pdf
Reference12 articles.
1. Wang T, Qin Y, Zhang J, Li X, Tong S, Zhao W, Zhang J (2020) An antiviral drug-resistant mutant of hepatitis B virus with high replication capacity in association with a large in-frame deletion in the preS1 region of viral surface gene. Virus Genes. https://doi.org/10.1007/s11262-020-01787-9
2. Peiffer KH, Kuhnhenn L, Jiang B, Mondorf A, Vermehren J, Knop V, Susser S, Walter D, Dietz J, Carra G, Finkelmeier F, Zeuzem S, Sarrazin C, Hildt E (2018) Divergent preS sequences in virion-associated hepatitis B virus genomes and subviral HBV surface antigen particles from HBV e antigen-negative patients. J Infect Dis 218(1):114–123. https://doi.org/10.1093/infdis/jiy119
3. Buckwold VE, Xu Z, Chen M, Yen TS, Ou JH (1996) Effects of a naturally occurring mutation in the hepatitis B virus basal core promoter on precore gene expression and viral replication. J Virol 70(9):5845–5851. https://doi.org/10.1128/JVI.70.9.5845-5851.1996
4. Bruss V (1997) A short linear sequence in the pre-S domain of the large hepatitis B virus envelope protein required for virion formation. J Virol 71(12):9350–9357. https://doi.org/10.1128/JVI.71.12.9350-9357.1997
5. Rabe B, Delaleau M, Bischof A, Foss M, Sominskaya I, Pumpens P, Cazenave C, Castroviejo M, Kann M (2009) Nuclear entry of hepatitis B virus capsids involves disintegration to protein dimers followed by nuclear reassociation to capsids. PLoS Pathog 5(8):e1000563. https://doi.org/10.1371/journal.ppat.1000563
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